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Title of Journal: Biomech Model Mechanobiol

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Abbravation: Biomechanics and Modeling in Mechanobiology

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Springer-Verlag

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DOI

10.1007/bf02536363

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1617-7940

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Computational analysis of biomechanical contributo

Authors: Z Li C Kleinstreuer M Farber
Publish Date: 2005/11/04
Volume: 4, Issue: 4, Pages: 221-234
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Abstract

This paper evaluates numerically coupled blood flow and wall structure interactions in a representative stented abdominal aortic aneurysm AAA model leading potentially to endovascular graft EVG failure A total of 12 biomechanical contributors to possible EVG migration were considered The results show that after EVG insertion for the given model the peak AAA sacpressure was reduced to 142 mmHg 118 of plumen and hence the maximum von Mises wall stress and wall deformation dropped by factors of 20 and 10 respectively Thus an EVG can significantly reduce sac pressure mechanical stress pulsatile wall motion and the maximum diameter in AAAs and hence prevent AAA rupture effectively In the absence of endoleaks elevated sacpressure can still be caused by fluidstructure interactions between the EVG stagnant blood and AAA wall EVG migration forces vary from 14 to 7 N for different EVG geometries material properties and hemodynamic conditions AAAneck angle iliac bifurcation angle neck aortatoiliac diameter ratio EVG size aortouniiliac EVG and hypertension play important roles in generating forces potentially leading to EVG migration

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  2. On the definition and modeling of incremental, cumulative, and continuous growth laws in morphoelasticity
  3. A semi-stochastic cell-based formalism to model the dynamics of migration of cells in colonies
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  8. Elasticity–density and viscoelasticity–density relationships at the tibia mid-diaphysis assessed from resonant ultrasound spectroscopy measurements
  9. Mathematical modelling of the degradation behaviour of biodegradable metals
  10. Utilizing multiple scale models to improve predictions of extra-axial hemorrhage in the immature piglet
  11. The effect of compressive loading magnitude on in situ chondrocyte calcium signaling
  12. The emergence of extracellular matrix mechanics and cell traction forces as important regulators of cellular self-organization
  13. Altered mechanical state in the embryonic heart results in time-dependent decreases in cardiac function
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