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Title of Journal: Pituitary

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Abbravation: Pituitary

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Springer US

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1573-7403

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Pituitary tumor apoplexy in patients with Cushing’

Authors: Osamah J Choudhry Asad J Choudhry Elkin A Nunez Jean Anderson Eloy William T Couldwell Ivan S Ciric James K Liu
Publish Date: 2011/09/17
Volume: 15, Issue: 3, Pages: 428-435
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Abstract

Pituitary apoplexy in patients with adrenocorticotropic hormone ACTH producing tumors is a rare occurrence We report four patients with Cushing’s disease harboring ACTHsecreting macroadenomas who presented with pituitary apoplexy We report the endocrinologic and visual outcomes of these patients after emergent transsphenoidal surgery A retrospective chart review was performed in 4 patients who presented with pituitary apoplexy from hemorrhage into an ACTHsecreting pituitary adenoma The patient charts were reviewed for clinical presentation neuroimaging findings intraoperative surgical findings pathologic findings and postoperative endocrinologic and visual outcomes All patients presented with acute headaches nausea vomiting and visual loss from optic compression MR imaging demonstrated a hemorrhagic macroadenoma that was confirmed at surgery All patients underwent emergent transsphenoidal decompression within 24 h of presentation One of these underwent an additional craniotomy to resect residual tumor Postoperatively all patients showed significant improvement in visual acuity and visual fields with biochemical remission confirmed on laboratory testing Significant weight loss as well as resolution of diabetes and hypertension was noted in all cases All four patients remained in biochemical remission at their most recent followup visit mean 40 months range 24–72 months Excellent endocrine and visual outcomes can be achieved after emergent transsphenoidal surgery in patients with Cushing’s disease presenting with pituitary apoplexy Although the cure rates of nonapoplectic ACTH macroadenomas are generally poor higher rates of remission can be achieved in cases of pituitary apoplexy This may be partly due to the effects of tumor infarction


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Other Papers In This Journal:

  1. IGF-I assays: current assay methodologies and their limitations
  2. Erratum to: An endoscopic modification of the simultaneous ‘above and below’ approach to large pituitary adenomas
  3. Somatostatin receptor ligands in the treatment of acromegaly
  4. Incidence of Cushing’s syndrome and Cushing’s disease in commercially-insured patients <65 years old in the United States
  5. Primary hypothyroidism presenting as pseudoacromegaly
  6. Sellar meningiomas: an endocrinologic perspective
  7. Low frequency of cardniac arrhythmias and lack of structural heart disease in medically-naïve acromegaly patients: a prospective study at baseline and after 1 year of somatostatin analogs treatment
  8. A comparison of cabergoline and bromocriptine on the risk of valvular heart disease in patients with prolactinomas
  9. Sellar and clival plasmacytomas: case series of 5 patients with systematic review of 65 published cases
  10. Analysis of GNAS mutations in 60 growth hormone secreting pituitary tumors: correlation with clinical and pathological characteristics and surgical outcome based on highly sensitive GH and IGF-I criteria for remission
  11. Sellar plasmacytomas: a concise review
  12. Increased clinical symptoms of acromegalic arthropathy in patients with long-term disease control: a prospective follow-up study
  13. Two cases of Kallmann syndrome associated with empty sella
  14. Multiple head and neck tumors following treatment for craniopharyngioma
  15. Modulation of VEGF/Flk-1 receptor expression in the rat pituitary GH3 cell line by growth factors
  16. Acromegalic gigantism, physicians and body snatching. Past or present?
  17. Effectiveness of self- or partner-administration of an extended-release aqueous-gel formulation of lanreotide in lanreotide-naïve patients with acromegaly
  18. Adipsic diabetes insipidus in adult patients
  19. Pituitary gland and β-catenin signaling: from ontogeny to oncogenesis
  20. Differential diagnosis of ACTH-dependent hypercortisolism: imaging versus laboratory
  21. A novel variation in the AVP gene resulting in familial neurohypophyseal diabetes insipidus in a large Italian kindred

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