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Title of Journal: Pituitary

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Abbravation: Pituitary

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Springer US

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DOI

10.1002/jobm.3620280106

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ISSN

1573-7403

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Increased clinical symptoms of acromegalic arthrop

Authors: K M J A Claessen S R Ramautar A M Pereira J A Romijn H M Kroon M Kloppenburg N R Biermasz
Publish Date: 2013/01/24
Volume: 17, Issue: 1, Pages: 44-52
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Abstract

Arthropathy is an invalidating complication of acromegaly This arthropathy deteriorates radiographically despite longterm disease control However the clinical course and its relationship to the radiographic course are currently unknown We aimed to investigate the clinical course of arthropathy during followup and its relationship to radiographic progression in longterm controlled acromegaly patients Prospective followup study We studied 58 patients mean age 62 years women 41  with controlled acromegaly for a mean of 176 years Clinical progression of joint disease was defined at baseline and after 26 years by the Western Ontario McMaster Universities Osteoarthritis Index WOMAC and Australian/Canadian Osteoarthritis Index AUSCAN questionnaires for lower limb and hand OA respectively and performance tests Potential risk factors for progression were assessed The clinical course of arthropathy was related to the radiographic course On average hand and lower limb function deteriorated during followup despite large interindividual variations Joint pain was stable over time High levels of pain and functional impairment at baseline were related to clinical progression of hand pain and functional limitations High baseline BMI was a risk factor for functional deterioration in the lower limb The changes in symptoms and radiographic progression during followup were not related In treated acromegaly patients joint function deteriorates during prolonged followup despite biochemical disease control although there was interindividual variation Clinical and radiographic course of arthropathy were not related Therefore in clinical practice a combination of clinical and radiographic assessment is necessary to evaluate the course of acromegalic arthropathy


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  2. Erratum to: An endoscopic modification of the simultaneous ‘above and below’ approach to large pituitary adenomas
  3. Somatostatin receptor ligands in the treatment of acromegaly
  4. Incidence of Cushing’s syndrome and Cushing’s disease in commercially-insured patients <65 years old in the United States
  5. Primary hypothyroidism presenting as pseudoacromegaly
  6. Sellar meningiomas: an endocrinologic perspective
  7. Low frequency of cardniac arrhythmias and lack of structural heart disease in medically-naïve acromegaly patients: a prospective study at baseline and after 1 year of somatostatin analogs treatment
  8. Pituitary tumor apoplexy in patients with Cushing’s disease: endocrinologic and visual outcomes after transsphenoidal surgery
  9. A comparison of cabergoline and bromocriptine on the risk of valvular heart disease in patients with prolactinomas
  10. Sellar and clival plasmacytomas: case series of 5 patients with systematic review of 65 published cases
  11. Analysis of GNAS mutations in 60 growth hormone secreting pituitary tumors: correlation with clinical and pathological characteristics and surgical outcome based on highly sensitive GH and IGF-I criteria for remission
  12. Sellar plasmacytomas: a concise review
  13. Two cases of Kallmann syndrome associated with empty sella
  14. Multiple head and neck tumors following treatment for craniopharyngioma
  15. Modulation of VEGF/Flk-1 receptor expression in the rat pituitary GH3 cell line by growth factors
  16. Acromegalic gigantism, physicians and body snatching. Past or present?
  17. Effectiveness of self- or partner-administration of an extended-release aqueous-gel formulation of lanreotide in lanreotide-naïve patients with acromegaly
  18. Adipsic diabetes insipidus in adult patients
  19. Pituitary gland and β-catenin signaling: from ontogeny to oncogenesis
  20. Differential diagnosis of ACTH-dependent hypercortisolism: imaging versus laboratory
  21. A novel variation in the AVP gene resulting in familial neurohypophyseal diabetes insipidus in a large Italian kindred

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