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Title of Journal: Pituitary

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Abbravation: Pituitary

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Springer US

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DOI

10.1007/bf03320325

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ISSN

1573-7403

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Incidence of Cushing’s syndrome and Cushing’s dise

Authors: Michael S Broder Maureen P Neary Eunice Chang Dasha Cherepanov William H Ludlam
Publish Date: 2014/05/07
Volume: 18, Issue: 3, Pages: 283-289
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Abstract

MarketScan Commercial database 2007–2010 age 65 years was used CS patients were defined with ≥2 claims of CS diagnosis while CD patients were defined with CS plus a benign pituitary adenoma diagnosis or hypophysectomy in the same calendar year Annual incidence was calculated by dividing the number of CS or CD cases by the total number of members with the same enrollment requirement during the calendar yearsCS incidence rates per million personyears were 486 in 2009 and 395 in 2010 The lowest rates of CS were in ≤17yearolds and highest rates were in 35 to 44yearolds CD incidence rates were 76 in 2009 and 62 in 2010 The lowest rates of CD were in ≤17yearolds and highest rates were in 18 to 24yearolds The rates varied by sex 23–27 in males 98–121 in females In females lowest rates ranged 25–40 in ≤17yearolds and highest 167–272 in 18–24 year olds In males there were too few cases to report estimates by ageIn the first large USbased study the annual incidence of CS in individuals 65 years old was nearly 49 cases per million substantially higher than previous estimates which were based primarily on European data Using similar methods we estimated the incidence of CD at nearly 8 cases per million US population These estimates if confirmed in other epidemiologic databases represent a new data reference in these rare conditionsThis study was funded by Novartis Pharmaceuticals Corporation Maureen P Neary and William H Ludlam are employees of Novartis Pharmaceuticals Corporation Michael S Broder Eunice Chang and Dasha Cherepanov are employees of Partnership for Health Analytic Research LLC a health services research company paid by Novartis to conduct this research


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Other Papers In This Journal:

  1. IGF-I assays: current assay methodologies and their limitations
  2. Erratum to: An endoscopic modification of the simultaneous ‘above and below’ approach to large pituitary adenomas
  3. Somatostatin receptor ligands in the treatment of acromegaly
  4. Primary hypothyroidism presenting as pseudoacromegaly
  5. Sellar meningiomas: an endocrinologic perspective
  6. Low frequency of cardniac arrhythmias and lack of structural heart disease in medically-naïve acromegaly patients: a prospective study at baseline and after 1 year of somatostatin analogs treatment
  7. Pituitary tumor apoplexy in patients with Cushing’s disease: endocrinologic and visual outcomes after transsphenoidal surgery
  8. A comparison of cabergoline and bromocriptine on the risk of valvular heart disease in patients with prolactinomas
  9. Sellar and clival plasmacytomas: case series of 5 patients with systematic review of 65 published cases
  10. Analysis of GNAS mutations in 60 growth hormone secreting pituitary tumors: correlation with clinical and pathological characteristics and surgical outcome based on highly sensitive GH and IGF-I criteria for remission
  11. Sellar plasmacytomas: a concise review
  12. Increased clinical symptoms of acromegalic arthropathy in patients with long-term disease control: a prospective follow-up study
  13. Two cases of Kallmann syndrome associated with empty sella
  14. Multiple head and neck tumors following treatment for craniopharyngioma
  15. Modulation of VEGF/Flk-1 receptor expression in the rat pituitary GH3 cell line by growth factors
  16. Acromegalic gigantism, physicians and body snatching. Past or present?
  17. Effectiveness of self- or partner-administration of an extended-release aqueous-gel formulation of lanreotide in lanreotide-naïve patients with acromegaly
  18. Adipsic diabetes insipidus in adult patients
  19. Pituitary gland and β-catenin signaling: from ontogeny to oncogenesis
  20. Differential diagnosis of ACTH-dependent hypercortisolism: imaging versus laboratory
  21. A novel variation in the AVP gene resulting in familial neurohypophyseal diabetes insipidus in a large Italian kindred

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