Authors: Offer Erez Ilana ShohamVardi Eyal Sheiner Doron Dukler Asher Bashiri Moshe Mazor
Publish Date: 2004/07/09
Volume: 271, Issue: 4, Pages: 296-301
Abstract
The study population consisted of 192 SGA neonates with hydramnios 5515 SGA neonates with a normal amount of amniotic fluids 3714 appropriate for gestational age AGA neonates with polyhydramnios and 83763 AGA neonates with a normal amount of amniotic fluid A crosssectional population based study was designed between the four study groups Multiple logistic regression analysis was used to assess the contribution of these abnormalities and different risk factors to maternal and perinatal complicationsThe combination of hydramnios/SGA was found to be an independent risk factor for perinatal mortality OR 2055 CI 126–334 Congenital anomalies prolapse of cord hydramnios SGA and grand multiparity were also independent risk factors for perinatal mortality Independent risk factors for neonatal complications were prolapse of umbilical cord OR 413 95 CI 148–115 hydramnios/SGA OR 272 95 CI 181–407 chronic hypertension OR 245 95 CI 102–59 congenital malformations OR 193 95 CI 114–324 and SGA OR 147 95 CI 107–2 Significant independent risk factors for medical interventions during labor were fetal distress OR 19846 95 CI 4727–82527 GDM Class B–R OR 2122 95 CI 234–19225 GDM class A OR 464 95 CI 262–821 severe pregnancyinduced hypertension PIH OR 774 95 CI 235–2542 hydramnios OR 195 95 CI 13–291 hydramnios/SGA OR 184 95 CI 112–302 and malpresentation OR 156 95 CI 132–184The combination of hydramnios and SGA is an independent risk factor for perinatal mortality and maternal complications We suggest that the growth restriction of these fetuses is responsible for the neonatal complications while the hydramnios contributes mainly to maternal complications
Keywords: