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Title of Journal: Endocr

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Abbravation: Endocrine

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Springer US

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DOI

10.1016/j.autneu.2010.07.023

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1559-0100

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Interleukin18 enhances glucose uptake in 3T3L1 a

Authors: YiSheng Yang XiaoYing Li Jie Hong WeiQiong Gu YiFei Zhang Jun Yang HuaiDong Song JiaLun Chen Guang Ning
Publish Date: 2008/02/05
Volume: 32, Issue: 3, Pages: 297-302
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Abstract

In order to characterize the potential causative effects of interleukin18 IL18 on insulin resistance we measured glucose uptake in 3T3L1 adipocytes treated with mouse recombinant IL18 IL18 surprisingly enhanced rather than reduced insulinmediated glucose uptake in adipocytes Moreover IL18 could counteract the glucose uptake suppression caused by tumor necrosis factor α in 3T3L1 adipocytes The mechanism dissection showed that the IL18 upregulated phosphorylated Akt and downregulated phosphorylated P38 MAPK These findings indicated that the elevated serum IL18 levels in obesity and diabetes might be a compensatory response to insulin resistanceThis work was supported in part by the National Natural Science Foundation of China No30400216 ShanghaiSK Research and Development Fund 2003007t Shanghai Excellent Youth Doctor 2004 Shanghai Youth Star of Science and Technology 05QMX1430

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