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Title of Journal: J Endocrinol Invest

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Abbravation: Journal of Endocrinological Investigation

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Springer International Publishing

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DOI

10.1002/bate.200590040

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1720-8386

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Amiodaroneinduced thyrotoxicosis suggestive of th

Authors: Michel Lambert J Unger P Nayer C Brohet D Gangji
Publish Date: 2014/04/04
Volume: 13, Issue: 6, Pages: 527-530
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Abstract

Amiodaroneinduced thyrotoxicosis AIT is generally believed to result from increased hormonal synthesis related to the iodine overload Thyroid damage has recently been incriminated as a pathophysiological mechanism We report 3 cases of AIT associated with clinical and/or biochemical features consistent with thyroid damage This hypothesis was supported by a painful thyroid case 1 transient high serum Tg case 2 a transient case 2 or persistent case 3 hypothyroid phase and an undetectable technetium thyroid uptake during the hypothyroid period case 3 These clinical observations support the previous histological data indicating that thyroid follicular disruption might contribute to the pathogenesis of AIT

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