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Title of Journal: Eur Radiol

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Abbravation: European Radiology

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Springer-Verlag

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DOI

10.1007/s11182-013-9920-z

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1432-1084

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Relation of the ischaemic substrate to left ventri

Authors: Nicolas Mansencal Renaud Tissier JeanFrançois Deux Bijan Ghaleh Nicolas Couvreur Mario Rienzo Pascal Guéret Alain Rahmouni Alain Berdeaux Jérôme Garot
Publish Date: 2009/11/21
Volume: 20, Issue: 5, Pages: 1214-1220
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Abstract

Contrastenhanced cardiac magnetic resonance CMR for infarct sizing has been validated in large animals but studies and followup are restricted We sought to 1 validate CMR for assessment of myocardial area at risk MAR and infarct size IS in a rabbit model of reperfused myocardial infarction MI 2 analyse the relation between ischaemic substrates and subsequent left ventricular LV remodellingExperimental reperfused acute MI was induced in 16 rabbits Ten animals underwent crossregistered cine and contrastenhanced CMR and histopathology at day 3 for assessment of MAR and IS group 1 The remaining six rabbits underwent serial CMR for the study of LV remodelling group 2In group 1 mean IS was 127 ± 64 and 127 ± 69 of total LV myocardial mass on CMR lateenhancement technique and histopathology P = 052 r = 093 No significant difference occurred between CMR and histopathology for the calculation of MAR and IS/MAR ratio P = 018 and P = 017 whereas correlations were strong r = 092 and r = 095 In group 2 mean LV enddiastolic endsystolic volumes and LV mass were significantly increased at 3 weeks compared with measurements at day 3 P  001 Significant correlations between initial IS and the increase in LV enddiastolic volume r = 066 and the increase in LV mass r = 048 were observed as well as correlations between initial MAR and the increase in LV enddiastolic volume r = 070 and the increase in LV mass r = 037Comprehensive CMR provides accurate assessment of IS and MAR in reperfused rabbit MI Infarct size is closely related to LV remodelling Through the infarct size/MAR ratio this approach has great potential for assessing interventions aimed at cardioprotectionDr Mansencal was supported by a grant from the Fédération Française de Cardiologie Paris France Prof Garot was supported in part by a “Dotation à la Recherche par équipe” from the Maison du Cœur Paris France None of the authors has any conflict of interest to declare

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